2001 - march - paper 1 and 2

Discussion in 'MRCOG Forum' started by Guest, Feb 15, 2005.

  1. Guest

    Guest Guest

    march 1 2

    1 TFFFT TFTTT
    2 FFTFT TTfTT
    3 TTFTF TTTFT
    4 TTFTF FFFTF
    5 FFFTT FTTTF
    6 FFFTF TFFTT
    7 FTTFF ?TTTTF
    8 TTTFF FFFTT
    9 FFTTF TTTTF
    10 TFFFF TFTFF
    11 FFTTT TFFFT
    12 TTFTF FFFTT
    13 TTFTT TTTFF
    14 FTTFF FTFTF
    15 TFF?TF FT?TFF
    16 TTFTT FFTTF
    17 TFFfT FFTFF
    18 FFT?_F FTFTT
    19 TTTFT TFTFT
    20 TT?FTT TFTFF
    21 FFTFT FFTTT
    22 TT?_FT TTFTT
    23 FFTTF FTTTT
    24 FTFTT TFTTT
    25 FFTTT FFTTT
    26 TFFFT FTTFT
    27 FTTTT FFFFT
    28 TTTTF FTFTT
    29 TTFFT TFTTT
    30 FT?T?T?T FTFFF
    31 TTTFF TFFTT
    32 FTTTT TTTFF
    33 TFTFF TT?TFF
    34 FTTFT TTFTT
    35 TTTTF FTFTT
    36 FTFFF TTFTT
    37 FTFFF TTFTF
    38 TTFFF FTTFT
    39 TTFFT FTFFT
    40 FTTTF TFTTF
    41 FTFTF FFFFF
    42 TFFFF TTFTF
    43 FTFFT TFTFT
    44 TFTFT TTTTT
    45 FTFTT FTTTF
    46 TFTTF F?TFF?T
    47 TTTTT TTFFT
    48 FTFTT TFFTF
    49 TTFFF TFFTF
    50 TFFTF F?_FTF
  2. Hi Ruby,
    please please post the questions as well.it would be very very useful if u can do them please.
    Thank u
    Priya
  3. Shipra

    Shipra Guest

    Hi Ruby,
    In Q-45. paper-2 march 2001-- a shd betrue and 'c' and 'd' shd be false.Stroke vol is equal in both venrticles...and depends upon end diastoliv vol. Pls check.Thanx.
  4. Shipra

    Shipra Guest

    In q-50, paper-2 b is true and d shd be false. Syncytial knots are aggregation of syncytium itself on sides of villus...these are changes seen in placental ageing.
  5. Guest

    Guest Guest

    hi shipra
    reg 45 - ur right onthe left and right out put being equal... will get back onthe other sin a short while - just checking.
    reg 50 - u right on b beign true, what abt d - u sure they dont contain aggregates of cytotroph?
  6. Shipra

    Shipra Guest

    yes i just confirmed it in my o&g text..
  7. Guest

    Guest Guest

    no 33 - are neutrophils capable of replication at site of inflamn? or do they jsut attract other neutrophils to the site?
  8. Guest

    Guest Guest

    no 24 paper 2: E - tissue macrophages are derived from monocytes and not plasma cells, right?
  9. Shipra

    Shipra Guest


    They aren't capable of replication...they've their own line of origin( myeloid series) from which they are derived.
  10. Shipra

    Shipra Guest

    Agreed
  11. Shipra

    Shipra Guest

    46- paper-2 BE are true...just confirmed
  12. Guest

    Guest Guest

    thx for 46 - where did u gt that info though? been looking for it.

    also45 a and c are true
  13. Shipra

    Shipra Guest

    got it in googles...Q45 c- are u sure....can u pls explain a bit
  14. Shipra

    Shipra Guest

    paper-Q-22 Are u sure abt option 'b' Plasma cells are CHARACTERSTIC of acute infection...? as in any acute infection there's leucocytosis which is characterstic....not plasmacytosis...pls confirm
  15. Shipra

    Shipra Guest

    In paper-2 ,Q-24- 'c' is also false...tissue macrophages have protective role against foreign particle by way of phagocytosis ,they dont have any role in intracellular pathogens or pathogens which resist intracellular digestion( like typhoid,t.b.). only 'a' and 'd' are true.
  16. Shipra

    Shipra Guest

    Q-21 paper-2 'c' shd be false.primary oocyte is diploid
  17. Guest

    Guest Guest

    discussed with a cardiologist just now -
    45 a is false : because stroke vol is only the amt ejectedout from the ventricle and is the diffrence b/w vol at end of diastole and end of systole

    45 c: true: basedon starlings law - longer the durn of diastole, more the amt filled in from atrium to ventricle - larger blood vol ejected out
  18. Shipra

    Shipra Guest

    Oh thanks Ruby, nice explanation...u r doing a great job...i m sure u wud be benifitted by ur efforts :)
  19. Guest

    Guest Guest

    i am not sure abt characterisitic, but i looked at it the other way around, they are not charat og chronic infections.... what say u?

    thx for correctingme on 24 c and 21 c
  20. Guest

    Guest Guest

    with all the mistakes - and the pressure rising, i feel otherwiseon my preparation! wonder if it were wise to sit in march - just dont feel i am ready for it yet!
    but thx for ur good words, only trying my best, since there is no time, felt its best to ask experts (to cut it short for explanation)....also i wishi knew abt the existence of this forum earlier!!
  21. Shipra

    Shipra Guest

    Don't worry...everyone is sailing on the same boat ...just keep studying and make the most of these few days remaining...
    Anyways...paper-2 Q-27 'b' shd be true...are u sure about 'a' as I never came across that..
  22. Guest

    Guest Guest

    yes bronchus is true,so is adeno ca of cervix - mentioned in one of the mcq books previously released by rcog
  23. Shipra

    Shipra Guest

    Q-25 ,paper-2 'e' -- u marked it true... epiphyseal plates arent affected in osteporosis... it mainly affects spine and pelvis., femur neck. Long bones are rarely affected..and if affected then thge changes seen are endosteal resorption. Irregular epiphyseal plates are seen in rickets.Can you pls confirm??
  24. Guest

    Guest Guest

    u r right shipra, it doesnt affect epiphyseal plates. some how that answer is marked flase in my book here?? :?:
  25. Shipra

    Shipra Guest

    And in Q-31 paper-2 ,'a' is false. Stored blood is kept at 4 degree celsius and not -4 degree. also 'd' is false as in stored blood platelete dysfunction and degradation of some of the clotting factors occurs hence we give platelete concentrates in case of platelete deficiency but never stored blood.Pls check.Thanx.
  26. Guest

    Guest Guest

    hi, thx for correcting onthe temp bit. reg plt repalcement, u r right in what u saying, but it says it may be used and we do use it although its use is not preferential to PRP, what do u feel ?
  27. Shipra

    Shipra Guest

    Well u may be right...is the word MAY in any RCOG question always right..??? :idea: ... Anyways..ok in q-7 paper-2 'a' is true surely ( De swiet) and are u sure about 'b' as according to my text At birth Hba1- 85% and HbF is 15%...can u pls confirm?
  28. Guest

    Guest Guest

    abt 7 a - it says deoxygenated blood is better buffer - agreed, but we are talkig abt neonates at birth - and they have mainly fetal HB at birth which binds more avidly than adult hb - the buffering capacity is a function of the binding too...i then discussed this with a pediatrician (in uk) and he says, with this respect, this statement is false for neonates at birth. does desweit mention this abt fetal HB??

    not sure if rcog likes to ask for positive replies thru the "MAY" factor - but most questions are true - - :wink:

    reg, 7b- at birth more than 50percent i thought was fetal, infact abt 85%, whcih then changes over a period of 1 onth and then 4 months to adult HB - i am 90 percent sure on this, will get back with a mor solid info tomr. its past 1 am, will cach up tomr dear... pls leave allur doubts here, will answer themfirst thing in the morn!
    thx
  29. Shipra

    Shipra Guest

    Ruby,
    Thanx for the pains u r taking in this discussion. :) Have a sound sleep 8)

    7a- --yes they've mentioned in relation to HbF..and at birth,as u said , majority of Hb. is HbF so it shd be true
    7b- I agree with you.on this ..u r right...just found in ganong....sorry for the confusion.
  30. Shipra

    Shipra Guest

    In Q-2 paper-2 - 'a' shd be false and 'c' shd be true. In starvation endogenous proteins are catabolised leading to steady rate of eccretion in urine....and Glucocrticoid / ACTH administration increases uric acid excretion in urine.Pls check.
  31. Shipra

    Shipra Guest

    In paper-2 Q 15-- 'b' is false it actually inhibits glucagon secretion. And 'c' is true for sure ...this is known as gluconeogenesis.
  32. Shipra

    Shipra Guest

    In Q-12,paper-2 -- 'a' is true and 'e' is false. Muscle glycogen cannot give rise to free glucose in blood as they lack Glucose 6 phosphatase enzyme which is present in liver. And glucocorticoids cause glycogen deposition in liver....couldn't find whether they affect muscle glycogen...? Pls check.
  33. Shipra

    Shipra Guest

    Q-19,paper-2 'e' shd be false..pls check
  34. Shipra

    Shipra Guest

    In paper-2 Q-20 'b' can be true....they are talking of haemophilia which is also an X linked disease....and do u have any idea ab't 'e' ...Can u explain pls. thanx.
  35. Shipra

    Shipra Guest

    Q17 -paper-2 'a' is probably true....all 3 types of polymerases are involved in its synthesis from DNA...Can u pls confirm..??
  36. Guest

    Guest Guest

    hi, in paper 2:
    12 a - u r right
    and e - i am not too sure either

    19 e - it is false
    20 b - factor VIII is von willebrand right? if so then this factor is autosomal dominant
    20 e - it is sex linked (and they associating it with 1 - which is autosome, so false)
    17 a - i am not sure , which are the 3 polymerases?
  37. Guest

    Guest Guest

    In Q-2 paper-2 - 'a' shd be false and 'c' shd be true. In starvation endogenous proteins are catabolised leading to steady rate of eccretion in urine....and Glucocrticoid / ACTH administration increases uric acid excretion in urine.Pls check.

    u r right abt a - being false, but if Glucocrticoid / ACTH administration increases uric acid excretion in urine, then c should be false. pls verify
    thx
  38. Shipra

    Shipra Guest

    Paper-2 - Q2'c' is false...I agree with you ...perhaps I too needed sleep at that time :lol:

    Q20-b- factor 8 is antihaemophilic globulin ...def. of which gives rise to haempphilia. Von willebrand factor is not clotting factor..its actually a protein which is synthesised in megakaryocytes and endothelial cells and circulates in plasma in multimers of varying sizes,it actually froms a complex with factor 8 and protects it from degradation and also helps in platelete adhesion.So its not factor 8..
  39. Shipra

    Shipra Guest

    In paper-1 ,Q20- 'c' 'd' both are false.
  40. Shipra

    Shipra Guest

    In paper-1 Q47 - 'd' shd be false...its bactericidal....individual components are bacteriostatic but the combination is cidal..
  41. Shipra

    Shipra Guest

    In paper-1- Q49 - 'b' shd be false...as when we say xyz difference is significant;that means it is unlikely to be merely due to chance.
  42. Shipra

    Shipra Guest

    In paper-1, Q37 'c' shd be true... and in Q33 'e' shd be true.
  43. Shipra

    Shipra Guest

    Also paper-1 38'c' shd be true
  44. Guest

    Guest Guest

    will get back with answers ina short while - off to work. :arrow:
  45. Guest

    Guest Guest

    Q20-b- factor 8 is antihaemophilic globulin ...def. of which gives rise to haempphilia. Von willebrand factor is not clotting factor..its actually a protein which is synthesised in megakaryocytes and endothelial cells and circulates in plasma in multimers of varying sizes,it actually froms a complex with factor 8 and protects it from degradation and also helps in platelete adhesion.So its not factor 8..
    thx for correcting me and enlightening me :)

    In paper-1 ,Q20- 'c' 'd' both are false.
    agree

    In paper-1 Q47 - 'd' shd be false...its bactericidal....individual components are bacteriostatic but the combination is cidal..
    yes, u r right.

    In paper-1- Q49 - 'b' shd be false...as when we say xyz difference is significant;that means it is unlikely to be merely due to chance.
    its true for sure, and what u r saying also means the same thing

    In paper-1, Q37 'c' shd be true... and in Q33 'e' shd be true.
    33 e is true,
    37 c - i think u r right, wht sthe other one we confuse it with? thx for correcting me

    Also paper-1 38'c' shd be true - u r right, it is
  46. Guest

    Guest Guest

    Calymmatobacterium granulomatis.
    Its cause is a gram-negative intracellular bacillus found in mononuclear cells, C. granulomatis (formerly Donovania granulomatis) - THATS WHY I GOT CONFUSED
  47. Guest

    Guest Guest

    can u throw some light on answers to question 30 - paper 1 march 2001??
    my answers are FTTTT??????
  48. Shipra

    Shipra Guest

    In q-30 paper-1-- my ans are bcd. In ganong its written...a moderate constant level( chronic) of circulating estrogen exerts a negative feedback effect on LH secretion...whereas during the cycle an elevated estrogen level ( acute)exerts a positive feedback effect and stimulates LH secretion....so the preovulatory rise in estrogen paradoxically stimulates LH and FSH secretion. so the last ans shd be false as its not the increase in GnRH pulse frequency that stimulates gonadotropin secretion but its the preovulatory rise in estradiol that does it...
  49. Shipra

    Shipra Guest

    paper-1- Q29 - 'b' shd be false as naturally occuring estrogens like 17 B estradiol goes extensive first pass metabolism in liver when given orally hence are inactive orally.
  50. Shipra

    Shipra Guest

    paper-1,Q22- 'c' and 'e' shd be false. Hormones implicated in degenaration of corpus luteum are prostaglandins and oxytocin.

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